In December 2019, coronavirus disease 2019 (COVID-19) was first described in Wuhan,
China, in patients complaining of flulike symptoms.1 The virus was isolated and
identified as a new strain of coronavirus, now named SARS-CoV-2 (severe acute respiratory
syndrome coronavirus 2 ) . Cardiovascular complications of COVID-19 have received less
medical attention; nevertheless, the first cases of myocarditis in COVID-19 patients have
been reported,3, 4, 5, 6 and myocarditis has been recognized as the cause of death in
some covid 19 patients.7 Pathology usually is focal within the myocardium, but there is a
risk of arrhythmia as well as progression to fulminant heart failure and cardiogenic
shock.

Clinical presentation of SARS-CoV-2 myocarditis varies among cases. Some patients may
present with relatively mild symptoms, such as fatigue and dyspnea,4 , 5 whereas others
report chest pain or chest tightness on exertion.3 , 6 Many patients do deteriorate,
showing symptoms of tachycardia and acute-onset heart failure with cardiogenic shock.3,
4, 5 In these severe cases, patients may also present with signs of right-sided heart
failure, including raised jugular venous pressure, peripheral edema, and right upper
quadrant pain.10 The most emergent presentation is fulminant myocarditis, defined as
ventricular dysfunction and heart failure within 2-3 weeks of contracting the virus.8 The
early signs of fulminant myocarditis usually resemble those of sepsis: the patient often
presents febrile with low pulse pressure, cold or mottled extremities, and sinus
tachycardia.10 Myocarditis is an inflammatory disease of the heart characterized by
inflammatory infiltrates and myocardial injury without an ischemic cause.8 The most
commonly identifiable cause of myocarditis in the United States and other developed
countries is viral.9 , Arrhythmia is recognized as one of the possible clinical
manifestations of COVID-19 patients. One observational study of the clinical
characteristics of COVID-19 patients in Hubei, China, reported a 7.3% incidence of heart
palpitations among its 137 patients 13 Moreover, Wang et al reported that arrhythmia was
a cause of intensive care unit transfer in 44.4% of COVID- Coagulation and inflammatory
parameters are mildly altered in COVID-19 infection, whereas MIS-C is characterized by
laboratory evidence of a proinflammatory and procoagulant state